| A B C D E F G H I J K L M N O P Q R S T U V W X Y Z # |
Platelet Aggregation
MessagePerformed at BUMCP.
IMPORTANT NOTE: Performed Monday - Friday ONLY before 5:00 pm
AZ Locations - Call for STAT courier prior to collecting the sample, and time collection with the expected arrival time of the courier.
DO NOT TRANSPORT VIA THE TUBE SYSTEM.
Must be received at BUMCP within 1 hour of collection.
IMPORTANT NOTE: Performed Monday - Friday ONLY before 5:00 pm
AZ Locations - Call for STAT courier prior to collecting the sample, and time collection with the expected arrival time of the courier.
DO NOT TRANSPORT VIA THE TUBE SYSTEM.
Must be received at BUMCP within 1 hour of collection.
Test Code
PLTAGGR
Alias/See Also
Ristocetin Aggregation
CPT Codes
85576
Includes
Tests included in the panel:
| ADP |
| Collagen (Weak and Strong) |
| Epinephrine |
| Ristocetin (Weak and Strong) |
| Arachidonic Acid |
Preferred Specimen
3.2% sodium citrate - blue top tubes (2.7 mL each); Whole blood - DO NOT CENTRIFUGE
Instructions
Patient should be fasting for 8 hours prior to have their blood drawn.
Patient should not have taken Aspirin or Aspirin containing drugs for 7-10 days prior to testing.
Platelet counts below 100,000/mm3 may give variable results and should not be used.
Patient should not have taken Aspirin or Aspirin containing drugs for 7-10 days prior to testing.
Platelet counts below 100,000/mm3 may give variable results and should not be used.
Transport Temperature
Room Temperature
Specimen Stability
4 hours
Reject Criteria (Eg, hemolysis? Lipemia? Thaw/Other?)
| · Plasma and cells are separated |
| · Delivered by pneumatic tube |
| · Incorrect anti-coagulant |
| · Specimen past stability |
| · Underfilled or overfilled tube |
| · Specimens collected in expired tubes |
| · Clotted, Hemolyzed, Icteric and Lipemic samples |
| · Patient’s platelet count <100,000/ul |
| · Refrigerated, chilled on ice or frozen samples |
Setup Schedule
Performed at BUMCP Monday - Friday
Reference Range
Clinical Significance
Platelet aggregation is clinically significant in the detection and diagnosis of acquired or congenital qualitative platelet defects. The platelet’s ability or inability to respond to particular aggregating reagents is the basis for differentiating platelet dysfunction.
